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211. The adenosine diphosphate (ADP)-stimulated platelet aggregation (PA) was assessed by conventional aggregometry.

212. NSA had no effect on the platelet aggregation induced by AA.

213. Function of platelet; Anticoagulation system; fibrinolysis; The principles of blood transfusion.

214. The term of "aspirin resistance" usually refers to laboratory resistance. It can be identified by measuring thromboxane A2 or thromboxane-dependent platelet function.

215. Adenosine diphosphate ( ADP) was analyzed by reversed phase HPLC, and rabbit platelet aggregation was measured turbimetrically.

216. Platelet activation has a closed relationship with thrombogenesis in Stroke patients.

217. The activation of random donor platelet concentrates and platelets apheresis collections ( plateletpheresis ) during storage were studied.

218. The main adverse reactions were hematologic toxicity. The manifestations were : WBC, neutrophile granulocyte, blood platelet and hemoglobin all decreased, then was digestive canal symptoms.

219. Changes of platelet count (PC) and mean platelet volume (MPV) were observed in anesthetized open-chest dogs after coronary occlusion.

220. Activated platelets are the primary source of LPA in circulation, which in turn stimulate platelet aggregation.

221. CBC with platelet count, fibrinogen level, partial thromboplastin time, and ABO blood typing and antibody screen are obtained.

222. The blood coagulative system in three groups was evaluated by testing platelet count (PLC), Active part thrombin time (APTT), prothrombin time (PT), thrombin time (TT), FIBrinogen(FIB).

223. Objective : To discuss the influence and mechanism of action on platelet agglutinate - tion by Qidantian capsule.

224. Objective : To improve the method for isolating platelet - rich plasma ( PRP ).

225. Objective: To determine the amino acid composition and isoelectric point of porcine platelet - derived growth factor.

226. The D-dimer, fibrinogen, hematin, and blood platelet were measured in these patients before and after urokinase administration.

227. Splenectomy and newer agents to boost platelet production are also under study in chronic ITP.

228. Objective To observe the expression change of platelet membrane glycoproteins of patients with hemorrhagic thrombopathy, and to explore its possible pathogenesis.

229. Thrombocytosis is often associated with chronic blood loss and iron deficiency because of increased platelet production in the marrow reacting to continued consumption and loss.

230. Methods Recalcification test, hemolysis test and platelet adhesive test were conducted for compact silk membrane, polyporous silk membrane, bovine pericardium and collagen sponge.

231. Methods:Count the platelet by automated blood cell counter in vein blood and diluted fingertip blood and smear of blood check by microscope.

232. Probably the most common platelet function defect is the irreversible inhibition of thromboxane (which is necessary for platelet aggregation) caused by aspirin administration.

233. It is suggested that the platelet reaction can serve as a marker of agonal stab wound and the leucocyte reaction as a criterion for timing of wounds is not feasible.

234. Platelet disorders are either quantitative (thrombocytopenia or thrombocytosis) or qualitative (thrombocytopathy).

235. The results of activity analysis suggested that these two chimera proteins not only have antithrombin activities, but gain platelet aggregation inhibitory activities as well.

236. FG is prepared from platelet - rich plasma or by mixing concentrated fibrinogen solutions with thrombin.

237. Prostaglandin is the dilation of blood vessels and inhibit platelet function.

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238. Objective To investigate the effects of personal film intervention in the apheresis platelet donors on anxiety and pain caused by vein phlebotomy during donating.

239. AIM: To compare the efficacy and safety of clopidogrel on anti platelet aggregation with ticlopidine.

240. Results Naokang can inhibited the platelet agglutination induced by ADP and AA.