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31 The bile acid concentration in faecal water also decreased with increasing dietary calcium, and this was not influenced by dietary phosphate.

32 This was stimulated further by additional supplementation with phosphate, especially in the colon and faeces.

33 An additional reason that serum phosphate may fall is that it may shift into cells.

34 It also occurs in alcoholism, diabetic ketoacidosis, and in patients taking antacids which bind phosphate in the gut.

35 Finally, as discussed previously, phosphate should almost never be given to patients with renal failure.

36 Consequently, precipitation of luminal calcium by dietary phosphate should inhibit these effects.

37 This has led to a shift away from the traditional use of phosphate and potash.

38 These protective effects of calcium are not inhibited by a fourfold increase in dietary phosphate.

39 The second interferon-induced enzyme adds a phosphate group to a protein that is essential for the synthesis of new proteins to begin.

40 Phosphate positions on the DNA-ribbon backbone are shown as circles, and the bases as horizontal bars.

41 Primary metals production and phosphate fertilizer mining also have major environmental impacts.

42 The buffer base is composed of the anionic buffers which include bicarbonate, phosphate, serum proteins, and hemoglobin.

43 These include the development of tetany and the deposition of calcium phosphate in soft tissues.

44 Supplementary dietary phosphate decreased the fatty acid concentration only on the low calcium diet.

45 Organically farmed crops are not sprayed with any chemical pesticides and are not treated with nitrogen, potassium or phosphate fertilisers.

46 Fields will be surrounded by barrier strips to prevent phosphate, nitrate, and pesticide pollution of water-courses.

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47 Transferases are enzymes that catalyze the transfer of groups, such as amino and phosphate groups, between compounds.

48 These are impregnated with immobilised Iron Hydroxide which forms Iron Phosphate when in contact with phosphates and locks them up in the media.

49 We subsequently determined the soluble faecal concentrations of calcium, phosphate, fatty acids, and bile acids.

50 Tyrosine phosphate is found concentrated at points of cell-matrix and cell-cell contact in other cell lines studied.

51 We found significant negative correlations in faeces between soluble fatty acids and bile acids and insoluble calcium and phosphate.

52 Calcium supplementation increased the intestinal concentrations of insoluble phosphate, which indicates formation of insoluble calcium phosphate.

53 Experimental phosphate deficiency in humans may induce renal magnesium loss.

54 Part of this controversy results from the fact that patients respond variably to phosphate administration.

55 It is therefore impossible to predict with certainty what amount of supplementation will result in what increment in the serum phosphate concentration.

56 As shown earlier, taurine conjugated bile acids are not precipitated by calcium or calcium phosphate.

57 This includes dietary phosphate and calcium restriction as well as aluminum hydroxide antacids.

58 Increased dietary calcium increased faecal phosphate because the formation of insoluble calcium phosphate inhibits the absorption of phosphate.

59 Both in this situation, and associated with insulin and fluid therapy of diabetic ketoacidosis, phosphate moves rapidly into cells.

60 In a sense, phosphate is taken out of circulation in the Ecosphere, diminishing the prospects of more life.