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121. Similar studies need to be done to evaluate the role of the cholinergic nerves in the gastric response to intragastric ethanol.

122. Conversely, operations that divert bile away from the stomach will ameliorate dysplastic features induced by previous gastric surgery.

123. Likewise, the carcinogenic action of bile may be related to the duration of exposure of gastric mucosa to biliary carcinogens.

124. There is no simple method to assess proliferation in single gastric crypts.

125. Case 1 was the only patient who had gastric lesions, all lesions since have been ileal.

126. Gastrin is the designation given to a family of protein hormones produced by the mucosal cells of the gastric antrum.

127. Only in two cases was a difference of more than one grade in the extent of gastric metaplasia found.

128. Gastric metaplasia tended to occur less frequently in our patients receiving NSAIDs, but the differences did not reach statistical significance.

129. Thus, in addition to duodenal ulcer disease, H pylori eradication may also cure gastric ulcer disease.

130. The effect of chronic alcohol abuse on gastric acid secretion is not as predictable.

131. Morphologically, this barrier is provided principally with tight junctions between gastric surface mucous cells.

132. The lesion has been described as a volcano with central whitish discoloration projecting from an otherwise normal gastric mucosa.

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133. Finally Aluminium Hydroxide/Magnesium Hydroxide is administered orally, as a neutralizer of gastric hydrochloric acid.

134. Peptic ulcers are produced by the self-destruction of the gut wall by pepsin and hydrochloric acid in gastric juice.

135. There are no studies in the literature to date linking miners with an increased risk of developing gastric H pylori infection.

136. The relative complexity of gastric crypt anatomy compared with colorectal mucosa has discouraged its use as an experimental model in proliferation research.

137. Secondly, stimulation of capsaicin sensitive neurones in the stomach enhances the resistance of the gastric mucosa against experimentally imposed damage.

138. Dietary replacement of arachidonic acid by eicosapentaenoic acid results in an enhanced resistance to ethanol induced gastric damage.

139. The average excess mortality for gastric cancer among coal miners was 126 per million, with a range of 65-226 per million.

140. The study of gastric mucosal proliferation may have important clinical applications.

141. Previous gastric surgery was uncommon in all three groups and showed no significant difference.

142. Withdrawal from heroin, usually described in lurid nightmare language, is actually like a severe attack of gastric flu.

143. Only one patient was non-white. Previous gastric surgery was uncommon in all three groups and showed no significant difference.

144. There was no significant difference in gastric emptying between the two groups.

145. To determine the extent of gastric metaplasia, multiple biopsy specimens were collected from standardised sites of the duodenal bulb.

146. So far, no prospective study has been performed to investigate the natural history of gastric metaplasia.

147. The association between gastric histopathology and Helicobacter pylori was also investigated.

148. Histological examination of these areas at this time confirmed a chronic gastritis and atrophic gastric mucosa.

149. Mutoh etal showed that intracellular glutathione was mainly responsible for protecting against gastric cell injury induced by ethanol.

150. Data on the phospholipid composition in gastric mucosa differ, but there is general agreement on the predominance of phosphatidylcholine and phosphatidylethanolamine.