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61. Conclusion Monocyte proportion and erythrosedimentation are independent predictors of carotid atherosclerosis.

62. Studies substantiate that IMT correlates consanguineously with the presence of coronary atherosclerosis.

63. As atherosclerosis develops, the artery walls become thick and damaged and lose their normal inner lining.

64. Atherosclerosis is a multifactorial disease and inflammation mediates its all stages, from initiation to progression.

65. Objective To observe Tiaozhi capsules effect of protecting vascular endothelium, anti - atherosclerosis and discuss its mechanism.

66. Objective:This report is aimed to investigate the different effects of menopause on coronary atherosclerosis in patients with familial hypercholesterolemia and familial combined hyperlipidemia.

67. Objective To explore the correlation between hyperlipemia, hyperlipoproteinemia and unstable plaques of carotid atherosclerosis in ischemic cerebrovascular patients.

68. Background: Atherosclerosis is a chronic inflammatory - immune disease with enhanced oxidative stress.

69. Conclusion CMV plays an important role in the pathological process ofthe atherosclerosis and atherosclerotic cerebral infarction.

70. Objective:To observe the influence of Typha angustifolia L. on hyperlipidemia[sentencedict.com/atherosclerosis.html], atherosclerosis(AS).

71. Objective To observe the effect of poplar skin monounsaturated fatty acid(PSMFA) on the lipid profile of atherosclerosis in rats.

72. Carotid ultrasound is a safe, noninvasive and relatively inexpensive means of assessing clinical and subclinical atherosclerosis.

73. To analyze the inflammatory mechanism of pulsatile low shear stress-induced atherosclerosis (AS) of the common carotid artery in rabbits in vivo.

74. Objective : To investigate the relationship between hyperglycemia and carotid atherosclerosis ( AS ) in Tibetan population.

75. Miller has been studying the link between atherosclerosis (hardening of the arteries) and nanoparticles which calcify within the arteries.

76. It can occur in atherosclerosis, diabetes mellitus, or decubitus ulcer, and after severe Burns or frostbite.

77. The pathogenesy of atherosclerosis is complicated extremely. There are several theories to explain it from different point of view.

78. Consequently, macrophage biology will be a major focus of the Keystone Symposia meeting on Molecular Basis of Vascular Inflammation and Atherosclerosis.

79. Because a report in the Journal of the American Medical Association reveals that some Egyptian mummies show signs of atherosclerosis, hardening of the arteries.

80. We investigated the impact of antihypertensive drug to prevent CKD incidence in hypertensive patients with advanced atherosclerosis.

81. It can decrease the concentration of HDL-C thus plays an auxo-action for regression of atherosclerosis.

82. Here is occlusive coronary atherosclerosis. The coronary at the left is narrowed by 60 to 70 %.

83. The significant increase in plasm HCY and copper may contributed to atherosclerosis disease and thrombotic disease of grafts.

84. Oranges rich in vitamin C, vitamin P, can increase the body immunity and increases the flexibility of capillaries, reducing blood cholesterol, can prevent hypertension, atherosclerosis.

85. Objective To investigate the relation between carotid atherosclerosis and lacunar infarction in hypertensive patients.

86. Atherosclerosis is a chronic inflammatory disease in which immune mechanisms play a pivotal role.

87. LDL cholesteryl oleate as a predictor for atherosclerosis: evidence from human and animal studies on dietary fat.

88. Increases in work time are positively related to progression of carotid atherosclerosis in middle - aged men.

89. Three aortas are to demonstrate mild, moderate, and severe atherosclerosis from bottom to top.

90. Increases in high-density lipoprotein cholesterol (HDL-C) levels during statin therapy are associated with regression of coronary atherosclerosis, a review of four clinical trials has confirmed.