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31 Syncope can be categorized based on the causative mechanism ( See Table below ).

32 Vasovagal syncope ( VVS ) is a frequent clinic apsychia syndrome.

33 Further investigations will need to the extent of nitric oxide's role in vasovagal syncope.

34 Objective To explore the clinical characteristics, diagnosis and treatment of arrhythmogenic cardiac syncope and Adams-Stokes syndrome.

35 Causes of syncope include any process that transiently reduces cerebral perfusion.

36 The clinical major appearances included: tetany, carpopedal spasms, convulsion, epilepsy and syncope with seizures.

37 Conclusions— PEA cases had a significantly higher prevalence of syncope in their lifetime, with other correlates, including black race, that were distinct from VF cases.

38 However, in the diagnosis of attention should be given some special spirit, state of consciousness such as syncope, seizures, stupor, mole disease attack, the differential phase locked-in syndrome.

39 What therapies have been advocated for the treatment of vasovagal syncope?

40 Objeclive To study the hemodynamic and neurohormonal changes in patients with vasovagal syn- cope during the head-up tilt (HUT) table test the triggering mechanisms of syncope.

41 Postural hypotension and vasovagal syncope are the major benign causes of syncope.

42 But differential diagnosis should be done on some special spirit and conscious states, like syncope, eclampsia, stupor, hysterism(http://Sentencedict.com), and locked-in syndrome.

43 Syncope must be differentiated from epileptic seizures, although seizures due to brain hypoxia can occur in a syncopal episode.

44 What history and physical examination data help to risk - stratify patients with syncope?

45 Objective To improve the content of post - natal care quality and reduce the incidence of postpartum syncope.

46 Results The diagnostic results of 30 cases were 3 cases of cerebral syncope or symptomatic epilepsy, 9 cases of cardiogenic syncope, 18 cases of reflexible syncope and other syncope respectively.

47 Conclusion This data shows that there is impaired baroreflex function in patients with vasovagal syncope, and this may be associated with reduced baroreceptor sensitivity and autonomic dysfunction.

48 What therapies appear most effective for the treatment of vasovagal syncope?

49 What diagnostic testing data help to risk - stratify patients with syncope?

50 Syncope occurs in severe brain MRI: the fourth ventricle, basal cistern to expand, suggesting that the brain stem, cerebellar vermis atrophy.

51 Neurocardiogenic syncope is a form of dysautonomia characterized by an inappropriate drop in blood pressure while in the upright position.

52 Conclusion:Most of patients with unexplained syncope have VS. ISOHUT may increase the positive rate. Metoprolol is an effective drug in treatment of VS.

53 Results The commonest clinical manifestations of ESPC were headache, epilepsy, syncope, dysphrenia and neurosis.

54 Combining 4 patients of quinidine syncope after cardioversion, we studied preliminarily the applicable problem of quinidine after cardioversion.

55 Conclusion Functional sinus bradycardia may lead to syncope, dual chamber cardiac pacemaker implantation is an effective prevention method.