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1. All had oesophageal varices at endoscopy.

2. Gastric varices Gastric varices are not uncommon in cirrhotic patients with portal hypertension.

3. In particular, the presence of esophageal varices may contraindicate prophylaxis as it is associated with postoperative hemorrhage.

4. To observe the degree of gastroesophageal varices and bleeding ratio and death reason in the patients with PCT.

5. Prof. Sarin: Bleeding from gastric varices is a major cause of death in portal hypertension.

6. Conclusion The therapy of endoscopic ligation for esophageal varices bleeding gets high hematischesis achievement ratio, and the varices disappears after many times of therapy.

7. Sometimes, esophageal varices can rupture, causing a life - threatening condition.

8. Those retroperitoneal collateral circulation can resenable those periesophageal varices as lobulated masses.

9. Finding at least moderate hepatic fibrosis or gastroesophageal varices (GOV) at oesophago- gastroduodenoscopy (OGD) has been an indication for combined transplantation.

10. Portal hypertensive gastropathy and gastroesophageal varices are import ant factors upper gastrointestinal bleeding.

11. About one - third of people with esophageal varices will develop bleeding.

12. Esophageal varices develop when normal blood flow to your liver is blocked.

13. In a prospective study of 100 cirrhotic patients, Hosking etal found anorectal varices in 44% and haemorrhoids in 63% of patients.

14. Another postmortem study of three patients showed portal vein thrombosis with fresh thrombus extending from recently injected varices.

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15. Objective: To compare Sengstaken-Blackmore tube plus posterior pituitary to somatostatin for the treatment of large hemorrhage due to cirrhosis with esophageal and fundal varices.

16. Objective To observe the treatment effects of modified operations on great saphenous vein varices.

17. Objective: To observe and compare the effect of stilamin and Sengstaken-Blackmore Tube in the treatment of large hemorrhage due to cirrhosis combined esophageal and fundal varices.

18. METHODS: Serum ACE was measured by UV - spectrocolorimetry in 40 cases of liver cirrhosis with esophageal varices hemorrhage before and after sandostatin therapy and 20 healthy controls.

19. Objective To explore the surgical methods of great saphenous vein varices.

20. Objective: To compare the effect of Somatostatin and Sengstaken-Blackmore Tube in the treatment of large hemorrhage caused by cirrhosis combined esophageal and fundal varices.

21. There have been numerous reports of glue embolization after endoscopic cyanoacrylate (CYA) glue treatment of gastric fundal varices (GFV), with some cases fatal.

22. However, none of these methods has been proven precise enough to be able to select patients who are likely to have varices and these patients should be endoscoped with normal gastroscopy .

23. Objective To discuss the effect of the therapy of the portal hypertension complicated with thoracic esophageal varices and bleeding by the esophagogastric devascularization.

24. The up GI X - ray examination revealed that esophageal varices was significantly attenuated 1 month after surgery.

25. Methods Clinical materials of laser treatment of 420 patients (525 limbs) with greater saphenous vein (GSV) varices were studied retrospectively.

26. ObjectiveTo determine the effectiveness of timolol in preventing first variceal hemorrhage in portal hypertensive patients with esophageal varices.

27. Objective To observe the curative effect of ligating esophageal varices under endoscope.

28. Results:The types of the cardia diseases included carcinoma (21), esophageal varices (14), mesenchyme tissues neoplasm(11) and esophagus hiatus hernia (8).

29. Objective To investigate the values of multi-slice spiral CT portography(MSCTP)in evaluating the embolization of gastroesophageal varices preoperatively.

30. In this study, there were 127 patients with esophageal varices hemorrhage. 62 patients were treated with Sandostatin, the others 65 with pituitrin.