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61. Objective To study clinical and pathological features of multifocal glioma and multicentric glioma.

62. Objective To investigate the expression patterns of Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) in glioma and its clinical significances.

63. Objective In order to observe the effects of phenylace te (PA) on cells morphogeny during the process of inducing differentiation of glioma cell line C6.

64. Glioma is the most common tumor in brain neoplasms, and also a difficult problem in neurosurgery.

65. Objective To discuss the etiological relationship between brain trauma and glioma.

66. Glioma cells with mutation in a specific gene called the phosphatase and tensin homolog gene, or PTEN, seemed more resistant to the immune system than glioma cells with normal PTEN function.

67. Results There were 7 cases with anaplastic astrocytoma, 3 cases with glioblastoma multiforme, 1 cases with oligodendroglioma, 1 cases with lower grade glioma.

68. Objective: To evaluate the result of brain lobe resection of frontal temporal occipitotemporal lobe glioma combined with postoperative radiotherapy and chemotherapy in 10 patients.

69. Results There are many pseudopodia and microvilli on the glioma cell sur face.

70. Glioma is the most common type of primary intracranial tumors in human. It developed from astrocytoma, oligodendroglioma, ependymoma, which were induced by neural ectoblast.

71. AIM: To investigate the effects of glioma cells on aquaporin expression in blood-brain barrier and their importance in pathophysiology.

72. Conclusion: MRI is an effective method in diagnosis of hemangioblastoma, however, the atypical tumors should be differentiated from glioma, simple cyst, meningioma and AVM.

73. Purpose To research the aspects of CT and MRI of optic glioma.

74. Sniggers Seizures and headaches are common first signs of a cleome glioma.

75. Purpose : To evaluate the value of MRI in diagnosis and differential diagnosis sylvian fissure glioma ( PSFG ).

76. The edema induced by glioma cells may not be the result of hyperpermeability of blood CSF barrier to macromolecules in plasma.

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77. But the sign can not be found in all 32 cases with intra-cerebral glioma, including 21 cases with astrocytoma and 4 cases with oligodendroglioma and 7 cases with ependymoma above cerebellar tentorium.

78. Conclusion: Matrine can induce C6 glioma cell apoptosis and the mechanism might be related to a number of genes involved in death receptor pathway and mitochondrial pathway.

79. Objective To study the quality parameters of epithermal neutron for boron neutron capture therapy and to obtain the optimized neutron beam used in human glioma therapy.

80. The location of the cleome glioma must also be considered when deciding treatment.

81. These lesions include 6 meningioma, 4 glioma, 2 inflammatory granuloma, 3 hemangioblastoma, 1 lipoma, 1 trigeminal neurofibroma and 1 parasite infection.

82. Objective To explore the changes in apoptosis level and proliferative activity of the C6 brain glioma cells after cryotherapy in rats.

83. Objective To investigate the invasion ability and collagenolytic activity of human glioma cells in vitro.

84. High - grade glioma is a common primary brain tumor with bad prognosis.

85. At low power, a glioma at the left shows greater cellularity and pleomorphism than adjacent brain at the right, but the margin is not distinct.

86. Conclusion: Downregulation of alpha B - crystallin mRNA may be concerned with the development of human glioma.

87. Objective To investigate the effect of bradykinin (BK) on tight junction (TJ) of the blood-tumor barrier (BTB) and interendothelial TJ of capillaries in brain glioma rats.