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(61) Helicobacter pylori is recognised as a significant cause of chronic antral gastritis and important in the aetiology of peptic ulceration.

(62) There are no studies in the literature to date linking miners with an increased risk of developing gastric H pylori infection.

(63) One of the H pylori negative patients was on maintenance haemodialysis and eight were on continuous ambulatory peritoneal dialysis.

(64) H pylori infection was proved by positive cultures or histological identification, or both in antral specimens from 113 patients.

(65) In conclusion, our findings suggest that H pylori infection may have been predominantly acquired at a young age in the past.

(66) Seven of the parents were positive for H pylori on urease testing, histology, and on culture.

(67) The association between gastric histopathology and Helicobacter pylori was also investigated.

(68) The H pylori negative and positive healthy volunteers were similar for both basal and peak acid output.

(69) Patients were divided into three groups according to the presence of histological gastritis and evidence of H pylori infection by at least one criterion.

(70) Atrophy, which is closely related to H pylori infection, is associated with a decline in acid secretion.

(71) Taylor suggests that in more than 50% of cases of acute H pylori infection,(sentencedict.com) hypochlorhydria lasts for several weeks.

(72) Of the nine H pylori negative uraemic patients, two had oesophagitis and one had scattered petechiae in stomach and duodenum.

(73) This finding provides support for the belief that adequate treatment of H pylori infection will give longterm protection from duodenal ulcer recurrence.

(74) These findings support the hypothesis that H pylori colonisation is directly involved in the hypergastrinaemia.

(75) The high pepsinogen C concentrations in patients with duodenal ulcer are probably related to H pylori related chronic active gastritis.

(76) The primary aim of the second trial was to see if eradication of H pylori led to resolution of intestinal metaplasia.

(77) Omeprazole monotherapy merely suppressed bacterial colonisation, especially in the antral region, and eradicated H pylori in individual cases only.

(78) Discussion Our current results confirm that treatment which both heals duodenal ulcers and eradicates H pylori significantly decreases basal plasma gastrin concentrations.

(79) H pylori infection has been shown in prospective epidemiological studies to be a risk factor for gastric cancer.

(80) It is concluded that successful eradication of H pylori improves secretion of vitamin C into gastric juice.

(81) The dynamics and importance of H pylori reduced altered mucosal peptic activity is far from being clarified.

(82) It may be secondary to the recent findings of reduced somatostatin concentrations in the presence of H pylori infection.

(83) This study aimed to analyse H pylori infection within family groups.

(84) The mechanism of hypergastrinaemia associated with H pylori infection is not known but the accompanying antral gastritis might be involved.

(85) One explanation for this finding would be that H pylori infection is acquired by people throughout their lives.

(86) After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.

(87) The one parent who was H pylori negative has histological evidence of mild inflammation.

(88) To evaluate our first impressions we examined transmission electron microscopy findings in 40 H pylori positive cases out of 94 randomised cases.

(89) Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.

(90) H pylori infection was a strong predictor of ulcer recurrences.