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(1) Only a minority of patients infected with H pylori will eventually develop a duodenal ulcer.

(2) Thus H pylori infection combined with active ulceration may cause the increased basal acid secretion seen in duodenal ulcer disease.

(3) It is now generally acknowledged that Helicobacter pylori infection is the major cause of antral gastritis.

(4) Results Eighty three patients with H pylori positive gastric ulcers entered the study.

(5) The Pyloriset detects IgG antibodies by agglutination, using Latex particles coated with acid extracted antigen of H pylori.

(6) It has also been proposed that H pylori ammonia production will lead to mucosal damage by denaturing the protective mucus layer.

(7) After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.

(8) H pylori was identified by culture and histology as previously described.

(9) The studies were designed to evaluate the H pylori eradication potency of the various regimens and the post-therapeutic course of ulcer disease.

(10) Most investigators have observed that H pylori infection causes a greater percentage increase in the postprandial gastrin than fasting gastrin.

(11) The present study indicates that H pylori infection is not the unknown factor.

(12) H pylori was not examined because its importance in duodenal ulcer disease was not widely recognised when this study was being planned.

(13) The high rate of H pylori ammonia production in uraemic patients should accentuate any ammonia induced effects.

(14) In conclusion, we investigated the influence of Helicobacter pylori colonisation on gastric mucosal eicosanoid synthesis in patients taking NSAIDs.

(15) Bismuth has also become popular in recent years as a treatment in peptic ulcer to eradicate Helicobacter pylori.

(16) Future studies evaluating mechanisms of gastrin release have to take into account the H pylori state.

(17) The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

(18) In theory the increased prostaglandin synthesis seen with Helicobacter pylori might explain such a reduction in minor mucosal injury.

(19) There have been conflicting reports concerning the form of gastrin that is increased in H pylori infection.

(20) There are few data concerning the role of anti- H pylori treatment in gastric ulcer disease.

(21) Meretek, a Nashville firm that manufactures the test, claims it is 95 percent accurate in detecting H. pylori.

(22) Increased basal serum gastrin is related to both atrophy and H pylori infection but not to ageing perse.

(23) One month after completion of this treatment their C-urea breath test was repeated to assess the H pylori state.

(24) Therefore, additional evidence clearly pointing to a causal relation between H pylori infection and gastric ulcer disease has to be provided. Sentencedict.com

(25) This would show that acid secretion is of superior importance compated with H pylori.

(26) The fall in basal acid secretion found may not necessarily be due to the eradication of H pylori.

(27) We have shown that the serum pepsinogen-I: -II ratio is significantly lower in H pylori positive subjects than in uninfected persons.

(28) It therefore seems justified to recommend amoxicillin/omeprazole as the treatment of choice to eradicate H pylori in H pylori related gastroduodenal diseases.

(29) On logistic regression, the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.

(30) The results of gastric juice against plasma concentrations according to H pylori status are shown in Figure 3.