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61. Psyllium significantly delayed gastric emptying from the third hour after a meal.

62. This increase in gastric secretion showed a positive correlation to the total number of cigarettes smoked.

63. Gastric emptying tests with radiolabelled meals and gamma camera imaging have proved popular for many years in clinical research and physiological studies.

64. Patients with previous oesophageal or gastric surgery were excluded from the study.

65. The stomach was then removed and the number of gastric lesions were counted.

66. The excess for gastric cancer was noted only for men over 70 years of age.

67. The experience of partial gastrectomy indicates that the time required for de novo growth of gastric carcinoma is about 15 years.

68. In addition, our data indicate genetic heterogeneity of gastric ulcer disease.

69. The normal gastric mucosa can prevent bypass diffusion of potentially noxious substances from the gastric lumen.

70. The patients with gastric ulcer had significantly fewer gap junctions than did the healthy volunteers.

71. Discussion A decrease in the incidence of gastric carcinoma has been widely reported over the last three decades.

72. Fasting gastric electrical control activity was recorded and analysed using a modification of the method described by Van der Schee etal.

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73. This observation illustrates the usefulness of endosonography in the staging of low grade gastric lymphoma.

74. On logistic regression, the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.

75. If these results can be confirmed, chemotherapy without gastrectomy should be the treatment of choice for agressive gastric lymphomas.

76. These reports suggested that inhibition of prostaglandin synthesis was unlikely to be sole mechanism responsible for the gastric damage induced by indomethacin.

77. Acid output was calculated by multiplying the hydrogen ion concentration by the volume of the gastric aspirate.

78. Neuropeptide Y has also been reported to increase substantially gastric acid and pancreatic exocrine secretion.

79. There are no studies on the acute effect of alcohol intake on gastric acid secretion in chronic alcoholic patients.

80. A recent review confirmed the increase in risk of developing a gastric carcinoma after gastric resection.

81. The results of gastric juice against plasma concentrations according to H pylori status are shown in Figure 3.

82. An increased risk of developing gastric cancer after previous vagotomy has also been reported.

83. In conclusion, we investigated the influence of Helicobacter pylori colonisation on gastric mucosal eicosanoid synthesis in patients taking NSAIDs.

84. In accordance with Wyatt etal we found a significant association between the extent of gastric metaplasia and microscopic signs of duodenitis.

85. Primary small cell gastric lymphomas are less frequent and are usually localised, solitary lesions that can be completely excised.

86. In addition, the phospholipid composition of gastric mucosa was compared with that of duodenal mucosa in 10 patients with duodenal ulcer.

87. The animals were anaesthetised three hours later and gastric blood flow and area of gastric lesions were measured as previously described.

88. The concentration of other essential cationic metals in gastric juice did not differ between healthy subjects and those with peptic ulcer disease.

89. Over the years there have been many studies of the effects of smoking on gastric secretion.

90. The protein was further shown to inhibit cAMP release of gastric fundic biopsies and to prevent its stimulation by histamine.