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1. In both patients naloxone led to great improvement, supporting the idea that endogenous opiates are involved in the condition.

2. Objective:To investigate the therapeutic effect of naloxone on ischemic hypoxic cerebropathy in neonates.

3. New application development of Naloxone in emergency and critical disease was discussed.

4. Conclu. sion : Naloxone can protect or improve the immunologic function in aluminium - poisoned mice.

5. In addition, the withdrawal symptoms precipitated by naloxone in morphine dependent rats were blocked by methoctramine(ip) or pirenzepine (it) at single dose injection in a dose dependent manner.

6. Conclusion High-dose Naloxone treatment on intoxation of hypnotic and alcohol is more effective than low-dose.

7. Objective To investigate the effect of naloxone on neurologic protection after brain trauma.

8. AIM : To evaluate the instant effect of naloxone for brain infarction with somatosensory evoked potentials ( SEP ).

9. To evaluate the instant effect of naloxone for brain infarction with somatosensory evoked potentials ( SEP ).

10. AIM: To observe the efficacy of naloxone treatment of primary brain-stem injury.

11. Methods:Summarizing the usage of naloxone in acute intoxation, acute cerebral infarction, acute respiratory failure, critical craniocerebral injury and shock.

12. Conclusion Naloxone treatment of joint meclofenoxate good effect in severe brain injury, it is worth promoting in clinical use.

13. In particular, those injected with naloxone did no better than the o ther two control groups.

14. AIM: To investigate the effects of tanshinone and naloxone on regional myocardial blood flow (RMBF) during ischemia and reperfusion.

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15. Objective To explore the role of naloxone in treating vertebral - basilar artery blood insufficiency.

16. Objective To observe the effectiveness of naloxone treatment for acute alcohol toxicosis.

17. To explore the influence of Naloxone Hydrochloride injection on T - AOC in acute alcoholism rats.

18. Objective : To study the application of naloxone in acute and critical diseases.

19. In such cases, drugs such as naloxone and naltrexone provide molecules that bind to opioid receptors with a higher affinity than the opioids themselves, but do not activate the receptors.

20. Objective To investigate the effect of naloxone in the treatment of acute organophosphorous pesticide poisoning.

21. Conclusion:Naloxone can improve the conditions of ischemic hypoxic cerebropathy in neonates to a definite degree and facilitates recovery.

22. Objective : To observe the curative effects of naloxone therapy on primary apnea of premature infants.

23. It has been observed in rabbits that hypotension caused by haemorrhagic orendotoxic shock could be reversed by injecting naloxone, phentolamine or cinanserineinto the lateral cerebroventricle.

24. Objective: To explore the leu-enkephalin(L-ENK) levels in the plasma and CSF of patients with cerebral hemorrhage, and the effect of naloxone on the levels.

25. Objective: Observe the curative effect of acute intensive alcohol poisoning by using naloxone hydrochloride.

26. Moreover, the potential analgesic mechanism of cinobufagin injection was investigated by injecting naloxone hydrochloride into mice.

27. Methods:The firing of ventral pallidal neurons in the action of morphine and naloxone was recorded and analyzed in the morphine dependent rats with extracellular single-unit recording.

28. The study suggested that elevated levels of endogenous opiate peptides in blood and CSF were involved in pathogenesis of epidemic encephalitis-B and naloxone is an effective therapy.

29. Conclusion: To prevent the toxic encephalopathy must actively shock and use Naloxone to clean raw surface.

30. However, the analgesic effects of acupuncture and morphine were not reversed by naloxone microinjected into the amygdala or the mesencephalic reticular formation. 2 .