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1. The occurence of acute hepatic impairment with intravenous amiodarone does not necessarily preclude the use of this drug by mouth.

2. But the case for amiodarone is not proved, and it has been associated with a higher incidence of sudden death.

3. A retrospective study showed that amiodarone was associated with a better prognosis in patients with documented ventricular tachycardia on electrocardiographic monitoring.

4. Severe acute hepatitis immediately after intravenous amiodarone has been reported four times.

5. Amiodarone also acts as a thyroid hormone receptor antagonist.

6. Arrhythmia was finally controlled with amiodarone and digoxin.

7. In a subset of patients, this results in amiodarone - induced hypothyroidism or thyrotoxicosis.

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8. Objective To observe the efficacy of amiodarone in patients with malignant ventricular arrhythmia.

9. In this study, amiodarone use was associated with excess early and late all - cause and cardiovascular mortality.

10. Conclusion The amiodarone is an effective drug to treat the patient with tachyarrhythmia.

11. The acting time of Amiodarone was 8.111± 5.766、 6.900± 4.748 and 17.20± 12.28 ( day ) respectively.

12. To compare the safety and efficacy of amiodarone and cedilanid on the treatment of paroxysmal atrial fibrillation.

13. Methods: Propafenone and amiodarone were given intravenously to 20 patients with permanent pacemaker 3 months postoperatively in which 13 patients were with temporary pacing system.

14. ObjectiveTo compare the efficacy and safety of intravenous amiodarone versus propafenone for immediate cardioversion of re cent-onset atrial fibrillation.

15. Objective To describe the clinical features of amiodarone pneumonitis with Hypermastigote lung infection.

16. Objective To survey the effect of amiodarone for the prevention of postcardioperative rapid atrial fibrillation.

17. Amiodarone influences thyroid hormone secretion and metabolism in all patients taking the drug.

18. To compare the safety and efficacy of amiodarone and on the treatment of paroxysmal atrial fibrillation.

19. Objective To investigate the effects of amiodarone on transmural dispersion of ventricular effective refractory periods ( TDR ) in the normal and hypertrophic canine heart in vivo.

20. Objective To compare the effect of sotalol and amiodarone on exercise electrocardiography and hemodynamics.

21. Objective To about the effect of amiodarone for the treatment of atrial fibrillationAF.

22. Conclusion It is effective and safe that intravenous amiodarone in treatment of AMI with AF.

23. In those with non-sustained ventricular tachycardia during electrocardiographic monitoring treatment with low dose amiodarone has been shown to be effective.

24. Current preventive strategies include the use of beta-blockers and antiarrhythmic drugs such as amiodarone and sotalol.

25. These observational findings are in to earlier randomized trials of amiodarone and to be validated prospectively.

26. Methods 78 patients were sinus rhythm by oral quinidine or amiodarone or direct current cardioversion.

27. Methods 78 patients were converted to sinus rhythm by oral quinidine or amiodarone or direct current cardioversion.

28. Class III anti - arrhythmic drugs ( such as amiodarone ) act ia prolongation of the action potential.

29. OBJECTIVE To establish a method for the determination of amiodarone and desethylamiodarone in plasma by RP - HPLC.

30. Flecainide was used for prevention of paroxysmal atrial fibrillation (PAF) in 18 patients with frequent symptomatic attacks that were resistant or intolerant to quinidine, amiodarone, and so on.