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1. Occasional islands of regenerative mucosa were present.

2. These cells infiltrate into the colonic mucosa and are abundant during the active phase of colitis.

3. In some experiments the mucosa was completely devoid of mast cells.

4. Discussion Phagocytic cells are found in the intestinal mucosa; they are heterogeneous immunohistochemically in both normal and inflamed mucosa.

5. An increased permeability of the mucosa in patients with coeliac disease has previously been shown by Bjarnason etal.

6. The inflammatory cells were absent in the colonic mucosa of Crohn's disease patients examined in remission.

7. The normal gastric mucosa can prevent bypass diffusion of potentially noxious substances from the gastric lumen.

8. As a result, the inflamed duodenal mucosa may then become susceptible to acid and this may ultimately lead to ulceration.

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9. We showed that luminol enhanced chemiluminescence from colonic mucosa biopsy specimens is increased in patients with ulcerative colitis.

10. Daily assessment of the oral mucosa. High protein diet and nutritional drinks.

11. In addition, the phospholipid composition of gastric mucosa was compared with that of duodenal mucosa in 10 patients with duodenal ulcer.

12. An additional 32 biopsies from adults investigated for diarrhoea but with a normal jejunal mucosa on histology were selected as controls.

13. The vascular endothelium seems to produce superoxide in the inflamed mucosa, which would exacerbate tissue injury in ulcerative colitis.

14. Colonoscopy showed total colitis with superficial ulceration and oedematous polypoid mucosa extending into the terminal ileum.

15. The position of peak labelling was deeper in the antral crypts at position 61 compared with position 26 in body mucosa.

16. Protocol 2 studied the phenomenon of adaptive cytoprotection in response to mild irritation of the duodenal mucosa in the three experimental groups.

17. Activation of such immune mechanisms may hence reflect increased antigenic load caused by the immune mediated lesion in the gut mucosa.

18. This may partly explain the lack of change in the appearance of the duodenal mucosa on serial biopsy in this condition.

19. Prominent among the protective mechanisms that are controlled by nociceptive neurones is the microcirculation of the gastric mucosa.

20. Immunohistological investigation shows the same changes in the gastrointestinal mucosa as seen in the salivary glands.

21. Inhibition of prostaglandin synthesis by NSAIDs is the major established mechanism by which NSAIDs render the gastric mucosa vulnerable to mucosal injury.

22. These findings suggest that anaerobic bacterial activity has a protective effect on the ileal mucosa.

23. N-acetyl-5ASA is the major metabolite of 5ASA and the primary site of metabolism is probably the colonic mucosa.

24. This viewpoint is also corroborated by the brisk response of rectal mucosa to local gluten infusion.

25. The addition of indomethacin resulted in a significant decrease in the radioactivity ratio between ulcerated and intact mucosa.

26. In addition, evidence has been provided for an active transport mechanism in the human intestinal mucosa.

27. These biopsies were later investigated in a light microscope to confirm a normal mucosa.

28. Moreover, there are no data in healthy man regarding the contribution that the colonic mucosa makes to whole body protein synthesis.

29. For positive controls, sections containing rectal carcinoma and normal mucosa were employed and for negative controls the primary antibodies were excluded.

30. We also examined whether upward expansion actually occurred in the background colonic mucosa by ex vivo autoradiography.