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61. The high pepsinogen C concentrations in patients with duodenal ulcer are probably related to H pylori related chronic active gastritis.

62. Balri etal found that the metabolic clearance rate of G17 was lower in patients with duodenal ulcers than in controls.

63. Discussion Our current results confirm that treatment which both heals duodenal ulcers and eradicates H pylori significantly decreases basal plasma gastrin concentrations.

64. Kothary etal have reported that terminally extended forms of gastrin in conjunction with G14 are more prevalent in duodenal ulcer patients.

65. Each of these abnormalities will increase the exposure of the duodenal mucosa to acid and thus explain its ulceration.sentencedict.com

66. The difference between mean rates of secretion of acid and pepsin in control subjects and patients with duodenal ulcer is about 190%.

67. The phosphatidylethanolamine value was higher in duodenal ulcer and lower in chronic atrophic gastritis compared with the control group.

68. Kurata etal reported that duodenal ulcers were diagnosed 2.5 times more frequently than gastric ulcers in Los Angeles, California.

69. After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.

70. The most obvious explanation is that reflux of duodenal content into the stomach after operation is responsible.

71. Hence, we postulated that adaptive cytoprotection maintains a physiological equilibrium between duodenal mucosal resistance and luminal acidity.

72. In 38 patients endoscopy showed focal abnormalities or signs of diffuse inflammation of the duodenal bulb.

73. Five patients had additional selective gastric vagotomy because of excessive gastric acid or a history of duodenal ulcer.

74. Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.

75. The aim of our study was to find out if the more potent prokinetic drug cisapride could prevent duodenal ulcer relapse.

76. There was no significant difference in phospholipid composition between antral and duodenal sites in duodenal ulcer patients.

77. Lysolecithin was the smallest component in the duodenal ulcer and chronic atrophic gastritis groups.

78. Peptic ulcer is a convenient term which covers both gastric and duodenal ulcers.

79. If you have many whorls, few loops, patterned palms the chances are you will develop duodenal ulcers.

80. Objective:To study the operative treatment of duodenal diverticulosis.

81. Duodenal atresia:Double bubble and polyhydramnios.

82. Symptomatic duodenal diverticulitis is rare.

83. Conclusion: More attention should be paid to duodenal injury.

84. Drawing ( coronal view ) shows the locations of duodenal fossae.

85. Cysteamine can deplete tissue somatostatin (SRIF) and induce duodenal ulcers in rats.

86. The complications of duodenal ulcer that require surgical management are hemorrhage , perforation, obstruction, and intractability.

87. In summary, vascular aneurismal lesion should be included in the differential diagnosis of a duodenal submucosal tumor-like lesion.

88. The authors reported nursing of duodenal fistula patients treated with balloon catheter isolating digestive juice early operation repair.

89. In a duodenal ulcer, a long post parricidal interval of two to three hours offend exists before pain is apparent and it may then occur immediately before next meal.

90. Objective To evaluate the value of magnetic resonance cholangiopancreatography ( MRCP ) in detecting duodenal diverticulum.